May 8, 2025 - Epoch Times
06:26
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| Time | Text |
|---|---|
| Cancer is not homogeneous. | |
| The somatic mutation theory, which is the current theory on which a treatment is based, posits that you have a mutation in a single cell and that gives rise to a whole population of cells that look the same and have the same mutation. | |
| But the Cancer Gene Atlas has shown that that theory is completely wrong. | |
| Cancer cells are very heterogeneous so they're made up of very different populations of cells with different mutations and one of the populations is the cancer stem cell. | |
| So it's a sub-population of the cancer. | |
| So these are generally slow growing but they distinct in that they have the ability to divide indefinitely and grow indefinitely and can Change their characteristics. | |
| And so basically if you get rid of the fast dividing cells, which is the cancer, you're left with the stem cells which then become the roots which grow back to form the tumor. | |
| The proportion of stem cells can vary from 2% of the population to 70 to 80%. | |
| So the ones with a low percentage of stem cells are more likely to be cured. | |
| By chemotherapy and obviously the more stem cells there are you need agents which get rid of the stem cells and as I said conventional chemotherapy does not get rid of stem cells and radiotherapy is even worse because it stimulates their growth. | |
| The stem cells are the cells that propagate to form the cancer. | |
| So it's very interesting because now I'm thinking about how chemotherapy works and so it We won't get these very slow-growing cells, so it could get rid of sort of the immediate danger to your life, these fast proliferating things. | |
| But meanwhile, these stem cells just kind of continue doing their thing over the longer period of time and bring back, you know, I don't know, the same cancer, different cancer, I don't know. | |
| Yes, exactly. | |
| So it may be somewhat genetically different, but it would be somewhat the same cell type. | |
| left alone will proliferate and develop the tumor but you know one has to realize that for a tumor to be palpable it has to be you know something like a million million cells and so you know three four five years down the line you know if you have a tumor cell it takes that long to manifest now obviously the doubling time differs from different tumors but generally it takes a lot of Cell divisions | |
| before you get a palpable, detectable tumor. | |
| Well, so now the million-dollar question, how do you get rid of the stem cells? | |
| Yes, so there are a number of repurposed drugs that do it, and this has been well-established in the scientific medical literature. | |
| So people are going to think that this is hocus-pocus. | |
| But there's been a lot of research in stem cells. | |
| There's been a lot of research in oncology journals on stem cells. | |
| One of the most effective treatments to knock out the stem cell is the famous horse deworming medicine. | |
| Ivermectin? | |
| Really? | |
| I mean, I actually had a doctor, I think at the most recent IMA, before it was the FLCCC conference, a surgeon explained to me that she saw some anecdotal cases where ivermectin was actually helping people recover from cancer. | |
| And so she was looking to do more research in that area. | |
| So ivermectin, tell me how. | |
| Tell me how we know this. | |
| Yes. | |
| So people think that it acts on cancer because it's an antiparasitic drug. | |
| And that the cancer is a parasite. | |
| So that's not how it works. | |
| So they're different biochemical pathways. | |
| So ivermectin kills the parasite by targeting certain pathways in terms of neurotransmission in the parasite, but it acts on a whole host of other biochemical pathways. | |
| So it acts on, there's a pathway called Wnt, there's a pathway called Notch, Pathway called I-kappa B. So for reasons that are truly astonishing Ivermectin acts on a whole host of biological pathways distinct from the pathways it acts on parasites and in that way it interferes with the proliferation of cancer cells and it seems that it's very | |
| effective for dealing with stem cells because these stem cells have primitive pathways. | |
| And evolve from embryonic development. | |
| And these pathways are particularly targeted, particularly Wint and Notch and Hedgehog. | |
| These pathways are targeted by ivermectin. | |
| I mean, that's amazing. | |
| But you're telling me that this is actually already in the scientific literature? | |
| Absolutely. | |
| For how long? | |
| There's a reluctance for the oncology world to accept this because then... | |
| They have to admit that they're not completely correct and that they would have to use repurposed cheap drugs, which goes against their mantra. | |
| Here's the big question. | |
| Is there any research showing that there are no sort of interactive negative effects of, say, using an ivermectin with chemotherapy, for example? | |
| Yes, so that's a good question. | |
| Most of the data shows that these drugs act synergistically or additively. | |
| There's no downside. | |
| Just make sure the patient has a good diet. | |
| Avoid foods with high glycemic index. | |
| Avoid carbohydrates. | |
| Make sure they get good sleep and exercise and give them ivermectin. | |
| It's not a difficult concept. | |
| You may want to throw in some curcumin as well, a few other. |
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