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June 8, 2020 - Clif High
41:18
critical thinking - june 8 2020 #2 plan D
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Time Text
Hello, it's 1138 a.m. on the 8th of June 2020, year zero, and this is um I know how the internet works.
So this is the commercial part.
You're gonna have to sit through this little bit of uh or jump over it, uh self-serving um uh information.
So what I'm gonna do is to oops to plug the pure sleep stuff by telling you about the um fat shock that you saw in the uh title uh entrance thing there.
Where'd the there's one?
Uh so fat shock is actually legit, okay?
So the scientists will leave that, have come up with an understanding now of why anesthetics do what they do to humans and um uh other mammalian uh vertebrates or vertebrate um mammals, okay.
Um because what they've discovered is that anesthetics come on in and they realign your fats in the lipid layers in the uh neurons in your brain.
And so a quick little so if this was a neuron, and here's a little bit of fats, and they're all lined up like this normally, then what happens,
and so that's your normal state, and that's how your brain neuron normally works, and there's you know, there may be stuff um you know being transported back and forth and all of that kind of stuff around these little fat layers, right?
And these little fat, these little lipid um uh fat, they're not globules, they're plates more than globules.
Um get like they're lined up in a particular order, and that's your uh normal waking state, okay?
So normal waking state.
Uh and then they come along and they gash you.
They give you ether, they give you some of these other anesthetics.
Well, uh, you can have anesthetics that are gases, you can have anesthetics that are drugs, uh injected, uh eaten, yeah all different kinds.
And so there's always been this question of how the hell do they actually work?
And what it's been determined, at least so far in the uh the evidence being presented by this studies and experiments being done, and this is at um let's see, uh side.
Um I think it was just under anesthetics.
Uh anyways, uh psych.com or is it a dot org?
Uh I'll dig it out and I'll put it into the the description here.
Anyway, the um the article goes on to say that they discovered that anesthetics work by shocking all of these little fats there such that they take an entirely different arrangement and uh and go a bit squirrely.
And so it it in the process of going a little squirrely in all these cells and all these neurons, neuron cells, your your fat delays start doing stuff that they're not supposed to, and that's the state of that we're gonna that we define as unconsciousness here.
Unconscious, okay.
And so you're uncon when your fats are disrupted, and that's what anesthetics do.
And this is a really key kind of a thing to learn because it now tells us why, for instance, sleep happens, right?
And in the sense that sleep is the is your uh fat uh platelets within the neuron cell structures rearranging themselves in a natural organized fashion, unlike the anesthetic, where it comes along and bombs you and you're out.
Um it also means it also tells us something about potentially about the use of psychedelic drugs that have the ability to transport consciousness, that is to say disrupt consciousness Location in a universal geographic sense without disrupting the consciousness itself.
So in other words, the anesthetics come on in, and we have anesthetics coming on in, and they disrupt it, and you you're you become unconscious here, right?
Because your normal waking state is disrupted.
But you could have uh psychedelic drugs come on in and disrupt it as well in an entirely different way, and instead of becoming unconscious, your consciousness becomes transported somewhere else while still remaining intact, more or less, right?
Because psychedelic drugs do that, it's a more or less kind of thing.
The self isn't destroyed, consciousness isn't destroyed, it's not altered, but the parameters in which it operates are altered, and now we know why.
Because the at least potentially we know why.
One of the things here is that the that it's potentially that this arrangement of the of the uh fat platelets in here, maybe for uh psychedelic journals or journeys, they all go horizontal, so to speak, right?
Which is basically to say maybe they all have a specific vibration, and it causes your consciousness to go where the psychedelic journeys send you, which would we'll call hyperspace.
But so it gives us some interesting and very intriguing uh things to think about relative to the mind and mind activity and the brain and how those two are interrelated because the mind is not the brain and mind and the brain is not the mind, so uh but they do interact,
and so here we have now in understanding a better understanding of anesthetics and what they do to the the brain, and this hints at um other modalities that can also influence the brain in positive ways, negative ways, perhaps as well, for um uh altering the mind and consciousness and and basically what you do with it, right?
So so what I'm saying is that we now understand something a little bit more as to what's going on, and I bet you because of the way that I had researched it, I mean, like seriously researched over the course of uh um like seven solid months doing uh biochemical analysis and biopharmacological analysis and uh molecular biochemical analysis and molecular biopharmacology analysis in order to develop this pure
sleep product, which I finally have a bag of, by the way.
Uh, this is one of them.
I didn't want to ask the guys in at uh purebulk.com that produce these to send me one until now because it was always been on short supply, we've always been sold out, and so better people should get it than I should have a bag here because I can make my own, right?
The this is a formula I did I devised, and I've got all the material here to create it and so on.
And so this is a bag of it, it's a 60-day supply.
There's a there's a little spoon thing in here that gives you your measurement.
It's like a teaspoon and a half.
Um it and here's the thing, guys, you can use it in water because it has a constituent in here that protects the stomach and actually um invokes 70 to 80 percent of the effect without them, but it is best used with uh taken with fats.
Okay, so uh I mean I designed it to be used with um uh uh dairy products, yogurt, uh you know, full fat milk, uh this.
I I was trying to build up my muscles.
One of the things I was doing here was providing components within the um uh elements in the mix that that tend to promote human growth hormone to natural levels in a nice gentle fashion, along with um harmonizing with your digestion system, and it all worked off of the fat layer.
So when I was devising this, I'd first gotten down to about 138 pounds, maybe it's 138.2, something like that.
And that's fully clothed, and I'd lost mass amounts of muscle, uh, especially uh upper body strength.
And so I had to come up with something, and so when I was doing this, I needed knew I needed to pack on the weight, um uh and um uh you just got a delivery here.
Uh pack on the weight, and in order to do so, uh I had uh put it into um basically milk and half and half.
So it's quite a high fat mix.
But but now that I've bulked back up, the half and half's gone this last year, so I haven't used it, so it isn't necessary.
But this stuff works very well with the um fats in milk.
Uh it works very well with fats in all kinds of milk, nut milks as well.
I hear really good reports out of Hawaii with um macadamia nut milk.
And um, but it doesn't work well with hemp.
Okay, some of the seed milks uh have a componentry to um uh that uh it's just disharmonious.
So anyway, so this is pure sleep.
Uh you can get it at purebulk.com uh and just do a search.
I'll have a link for it.
This is the little commercial part in the video.
Uh so maybe you um watch through all of this.
Uh pure sleep is really good.
I've built back all kinds of muscle mass here.
I'm out being energetic, doing shit, causing problems for the world.
So, in that sense, it's been a very, very great recovery aid.
And I'm recovering from colon cancer and from the deprivations and scarring and trashing of the colon cancer surgery.
Uh so uh I needed all the help I can get.
There's no question about that.
And I'm doing okay with it.
I mean, I'm doing really damn well, actually.
So that was the the um the fat shock stuff, right?
So it validates my thinking about how uh those elements that I was after could aid sleep and then aid the body during sleep.
Because I discovered you know, quite factually, our bodies can do nothing more than triage for repairs um against damage while we're awake.
In other words, you only heal when you're asleep.
I could only build muscle mass when I'm asleep.
If you're out there, you know, tearing ass running or you're kickboxing or something all day long.
Your body's not making new muscle while you're doing that.
Your body's only making new muscle when you get your ass flat, you start relax, and this entirely different set of processes take over, right?
It's then that the human growth hormone home comes on out.
It's then that these home hormones are activated.
So, for instance, you go um uh out and get vitamin D, you get sunlight during the day, your body doesn't use it during the day, it's a steroid hormone itself, and your body uses it at night.
Same thing is true with melatonin and all of these kind of hormones, right?
Uh, it's I in my opinion, it's unwise to take the hormones directly, except in the cases of the vitamins.
Um, those kind of supplements are not issues.
So when I designed the pure bulk stuff, I had to take all this stuff into account, right?
Because I wanted to have a nice gentle sleep, especially with my colon cancer.
I didn't want it uh hassling with my digestion at all.
I wanted to wake up without any kind of uh hangover, I wanted to wake up with a happy feeling, didn't want it to be oppressive, I didn't want it to um uh alter the mind state to the point where uh you felt like uh a residual effect in any way throughout the day.
Uh I I didn't want it to combine with stuff like alcohol and produce um uh even seriously more effects, you know, that kind of thing.
So if you were a drinker, and so it it just does not um you know, so it was it was a lot of work anyway, and it and it works very well, uh specifically for what it does.
It's a gentle sleep-inducing aid that assists your body and provides your body with the nutrients that that it needs to accomplish those tasks while you're asleep.
And then you wake up feeling refreshed, there's no hangover.
I I have found uh we've had no one wake up and tell me that they felt bad as a result of taking this.
Uh had all kinds of people using it in um tests while I was um you know developing various different parts of the formula and so on.
And it's consistently worked, and I'm now it's over 19, it's probably like 21 months or something that I've been uh basically with this formula here.
We've got a new grind that I'm testing out now.
We have to do this with each batch that comes from a different supplier because we're dealing with Products that have aromatic uh components in them, like uh biochemically aromatic, that is to say they have specific uh chemical um constituents that can be affected by aging and oxidation and all this sort of thing.
So uh so we're and also with the stresses of the supply chains being disrupted, we have those issues as well.
But pure bulk has been very good.
We have some in stock, um, and we're working on the next batch, so things are proceeding quite well.
And lots of people say it it works for them within the first half hour, and I know the the I really screwed up last night because I was thinking, oh, I'm a little tired, I was kind of aching.
I had to repair my tractor, and uh, so I had some a little bit early, and it's like damn, out by 7 45, you know, and and stumble on into bed.
And but it was a great sleep.
So, you know, it compensates, yeah, I'm getting up really damn early, but I'm also getting stuff done.
So, okay, so on to other stuff here.
That was our our little commercial, but it's nice to be validated.
I was correct about the fats and the sleeping process.
Okay, so now the next thing to talk about is our disease issues relative to something else I was correct about.
Okay, so right at the moment uh we have India.
Let me get another.
Okay, so right at the moment we have issues in India.
These are clusters.
Uh uh SARS-CoV-2.
India, there's a small flush in Iran, it's small.
We also have Brazil.
And in all cases, these were um predicted from actual um theories developed from uh on the ground emerging data.
Okay, so there's a bunch of different ways you can do science.
One of the ways you can do science is you can um come up with a uh hypothesis, do testing, have it lead to a theory, uh, perform more extensive uh experimentation to attempt to falsify the theory.
If it can't be easily falsified, you adopt it as being at least operable, and so that's one branch of science.
Another way here is um science on the move, so to speak, where where it's basically an extension of uh survival um uh characteristics of our species, and that is you see shit happening, um, you react to the shit happening in the most logical way you can relative to the evolving situation on the ground,
and uh hope you're correct, but you don't stop and try and formalize the process until you get some understanding of what's going on, then you make predictions, and then you see if it plays out in the uh developing chaos around you.
If so, then you can rely on those predictions and that uh the underlying hypothesis that supported those predictions a little bit better as you go forward.
So uh we have the latter going on with this right here.
It was predicted based on uh vitamin D levels floating around through the population of the earth that we would have an outbreak in India when monsoon season hit.
We're now there.
India has a big outbreak.
India has uh monsoons that uh okay.
So the people in India, especially southern India, uh have darker skin, therefore it's harder for them to make the vitamin D. They have to have greater skin exposure, there's a cultural predisposition to not do that.
They want to have lighter skin, seeing it as cosmetically better.
Uh and so they don't stay in the skin or don't stay in the stun because it darkens their skin, so that's a the darkening of the skin is against their cultural predisposition, so they have a tendency to be low in vitamin D. Now, the poorer people are out in the sun a lot more, so they have a higher level of vitamin D than other people within their gen the more rich people within their general population, except in a period of time of the monsoons when everybody's covered by clouds.
And here's the thing about vitamin D. Vitamin D has a short half-life in your body.
The stuff you take by supplements, it's out of your body within four days.
Uh, if you don't, so if you took one supplement, uh basically they can't really measure it after about six hours, I think.
Uh, but it's known that it's half-life that you wouldn't have any residual effect uh four days later.
And vitamin D you make from the sun stays in your body about twice as long.
So but it's so if we're looking at a four-hour to sixteen-hour effective rate for a supplement, then vitamin D in the skin would be about a 24 to 30 hour effective rate of dosage, but it it would have some uh component in it in it still about eight days later.
But that's it.
So if you're under a cloud cover for two months, your vitamin D and you don't supplement, your vitamin D level is naturally going to be depleted because vitamin D is a steroid hormone and it's used continually in the body for signaling over 3,000 uh genetic processes, and so it gets used up.
And so here we have the in India we have the um uh outbreak now uh due to the monsoon season coming on.
This can be expected to also influence areas like Indonesia, Malaysia, Thailand, etc.
As we get into those areas, uh we'll see more of an outbreak of the COVID there.
Uh now in Brazil, we have um the fact that big chunks of Brazil are below 32 degrees, and it's only from uh 32 degrees north to 32 degrees south of the equator that you can make um that dark-skinned people,
especially dark-skinned people can make vitamin D. Um beyond 32 degrees uh you can white people can make vitamin D if the if the ultraviolet is there, but in the winter, even they can't make vitamin D because the ultraviolet is not there.
So uh so and really what we're looking at here with this cove thing is that it's a pandemic of it's a collision between the virus and a pandemic state of low vitamin D. So if you're low on your vitamin D and you run into the virus, you know, the outcome is not favorable.
But if you have vitamin D above 40 uh nanograms per milliliter, such people are not reporting to the hospitals.
That is to say, when they take blood draws on people going into hospitals, no matter where it is, India, Brazil, Sweden, wherever, those individuals that are going into hospital with uh the COVID 2, which means they've presented themselves there, they're symptomatic,
and they're they're obviously ill, requiring hospital uh care, they've got low vitamin D below uh 30 nanograms per milliliter, and uh if it's much below that, the outcome prognosis is very poor.
There I put a bunch of um, if you go to the half-pasthuman.com website, and you click on the blog and look at the list, you'll see a couple of uh uh blogs I put out there with video links for people that are discussing the vitamin D linkage, latitude, and so on uh in absolute detail.
So this seems pretty clear relative to the uh COVID 2.
Now Iran has a small outbreak going on, and it's because of their cultural conditioning and the fact that that a big chunk of the country is you know above 32 degrees.
And so even though it's getting into summer, because of the way they dress and so on, they're in a situation where they've got another round of it going through the population.
Basically, it would seem that on a logical expression only that if all humans took vitamin D to the level of at least 5,000 international units per day, we wouldn't have COVID 2 to deal with, and probably wouldn't have any coronavirus to deal with because the whole coronavirus shell appears to be uh vulnerable to the vitamin D and C. And so there's two issues there.
Okay.
There's the issues of receptivity and permissibility.
These are like virology terms that we're using them now, and so cells can be receptive.
And then they also need to be permissive.
All right, and so you've got a got your virus uh, okay.
so you have your cell.
And your cell has a couple of these Y guys.
And so these are the receptors that everybody's talking about.
A2.
And these receptors make that cell receptive to the virus.
Alright.
The virus can lodge on there and do its shit to get entrance into the cell.
So that's that's the component here.
But you need both of these components in order to be vulnerable to the virus.
And uh the second one is every bit as important.
So we have a situation here where uh lots of people would have ACE2 receptors, and so they would have receptive cells.
And this includes the Chinese, and the Chinese seem to have more ace two receptors in some places in their body than uh other racial groups, so they're more susceptible there, etc.
etc.
But here's the thing there's this other component, and that is the permissiveness, the permissibility of a cell.
And what that means is that after the virus gets its stuff in here, that's probably the wrong color for the virus.
Um, so anyway, after the virus gets its stuff into the cell, is there a protease that will allow that virus to replicate?
Because not all protease will allow all virus to replicate.
Either the constituent uh material isn't there, or the protease itself is not um hijackable because of uh one element or another in it.
So you have to have the the permissibility, it had that cell has to be able, it has to permit the hijacking of it by the virus, all right?
And so this is where the vitamin C and the vitamin D come on in.
Vitamin C and actually protects protects by electron donation, all right.
So it donates an electron and changes its chemical nature from uh from a pure vitamin C to something that's more of a like of an altered sugar, and we don't need to get into that, but it donates an electron in the process.
So when you have your uh virus critter come trucking along, and it's out here and it comes trucking along here, you can actually get immunity in your body that does not generate antigens, does not or does not generate antibodies from antigens.
So you wouldn't test for antibodies, but you could be a hundred percent immune to this because your cells are neither receptive nor are they permissive.
Okay, so in this case, vitamin C uh alters both of these.
Vitamin C is an electron donor.
As the virus comes on up here, you can get your body healthy enough with enough vitamin C in there, which basically means that like your batteries charged, it's prepared to donate electrons at the at the drop of a hat, and it doesn't matter because you're continually reflush ref refreshing it with vitamin C. And so what happens is that you get a um a negative charge effect against the virus from your body finding uh uh itself triggered
in an into an antiviral behavior, and it's triggered, and it doesn't give a shit what virus it is, just the fact that it is a virus causes a virus or a fungus or um a bacterium or an amoeba.
All of these can cause this effect with the vitamin C when they attack the cell, it can be an electron donation that literally can cause that the virus to be disrupted at its own fat layers and and at its own shell, should it have it, not all viruses do, um, and and disintegrate it.
So the donation of the electron can cause uh you know basically burst apart the virus to the point where it's not effective anymore.
Now there's something else about uh let me stop for a second and say something else about the um the or let we go back up here to the PFU.
Okay, we'll come back, we'll come back to that, all right.
And it doesn't mean pretty fucked up, all right?
But we'll come back to that.
Um so anyway, so here we are dealing with this.
So vitamin C can cause your cell to be not receptive, to come on up and say, nope, nope, nope, not gonna happen, right?
Uh, because it's gonna donate uh the electron and and disrupt the virus, and other things it can do is that the vitamin C can because it's part of the intercellular cement, the intracellular matrix that bonds the cells in here,
and it's part of the part of the cell wall maintenance structure right here, and because it's part of the protease structure,
it actually forms can be used by all of these, it can make your your cells not permit the virus to either enter or try or allow the protease hijack, and that's that's vitamin C on its own.
Now, you're likely not to supplement just with vitamin C, so you're gonna also have the vitamin D aspect of this as well, and that gets really kick-ass.
So, so here we have the vitamin C affects both receptivity and permissibility, and vitamin D, uh, which is as I say, a steroid hormone, it affects 3,000.
Um lots of which are involved in your immune system, and thus we see uh the link as to why uh virus and bacteria and this kind of you know influenza pneumonia and those sorts of things are uh prevalent when vitamin D is low, right?
So uh it doesn't mean that it causes it or any of that kind of stuff, but if you got low vitamin D, you're you're you have a high receptivity and a high permissibility, which means a high potential for becoming ill.
If you got adequate vitamin D, then your receptivity is low and your permissibility is low.
If you have adequate vitamin D and vitamin C, then basically you have very, very, very, very low receptivity and very, very, very low permissibility.
And it and it may be that you might consider yourself to be para-immune to to these kind of critters, even though you would test with no antibodies.
And there's probably a lot of healthy people out there like that.
Okay, so um, so that's that's the wrap on the aspects of why the vitamins, and I'm glad that I was proved correct that we have the India kind of a correlation, and we can plot this, we can see it developing in real time, and we can say, okay, four weeks from now it's gonna be monsoon season.
If there's a correlation in latitude, vitamin D, uh, skin color and and nutrition, etc.
etc.
etc.
Then we'll see a bloom or clusters develop here in India.
Damn if it didn't happen, okay.
That's yet again more proof that this thinking is correct, and that this whole COVID 2 thing is um uh latitude and vitamins um uh weak, okay.
And vitamin D is especially key for all your other healthy things anyway.
So you probably need to get um you know serious about taking the vitamin D. So now before we go into the other stuff, let's deal with the PFU.
There's something else.
Okay, so this whole COVID deal, right?
It's all evolving.
Everybody, there's new information coming in all the time, and so there's this concept, and and it doesn't mean pretty fucked up, but it there's a ratio, all right.
And this ratio is basically how infective, how infectious is a virus, all right?
And so viruses are strange critters.
So here you have your cell, here you have the virus come on in.
The virus replicates itself by hijacking your protease, and it does its stuff in here inside your protease.
And what it's doing Is that it's it's replicating itself because the virus is just genomic material.
Okay, it doesn't have any of the functions of a cell, it's not even technically alive when it's not in the cell, it certainly is uh alive when it's participating in the actions of the cell and stealing the stuff for its own replication, and at that point, what it's but it's replicating itself not like uh not like cells do by splitting in half and splitting uh meiosis and mitosis, it doesn't work that way, right?
The way it does it is it builds all these little constituent particles of itself, little little bits and pieces all around inside your protease, various different kinds of them, and then at some point it comes along and it tells because of triggers within itself when it knows there's a certain amount or whatever the conditions are,
it has this protease go over to the cell wall and do a thing they call lysing, which is to burst open the cell wall and spew all this shit out, heads harder than that, because it spews out all of this crap, right?
And it comes on out here, and you have a big bubble of this, and this um expulsion is actually what pollutes your body and makes you ill, and that your body really reacts to, and because it causes the body to trigger all of its um immune system stuff, right?
And so in this process, we discover something about the cove 2, and that is that when it does this, it has all of these little little particles, all right.
And so let's say that that this virus is composed of a little engine particle and uh um a little location particle and a little uh transport particle, and so it has to make these three chunks and it has to fuse them together to make a complete virus.
It makes all of the little bits in here, and then it it lyses the protease out of the cell and spews it out into the intercellular matrix where it's gonna assemble these critters here into a uh complete virus, and so these guys would all go in and they would make up our coronavirus when it was all done.
The thing is, though, it's only this, it's only the 100%, 100% pure coronavirus that can uh re infect another cell.
Now, the reason viruses are so deadly is that that one virus particle getting in here can create millions upon millions of virus particles because it's doing it in a way that's unlike the divide and divide and divide and divide, so it can go exponential, and that's really the the horror of viruses in general as as uh pathogens.
But all viruses, it's this is true of all viruses, all viruses fail repeatedly in making copies of themselves.
All right, so it's just true.
The viruses build these little uh genomic sections here, and then they have to do the assembly.
They fail in the in the in the building of the genomic sections, and then they fail in the assembly.
So there's lots of failures.
Mostly, and this is why they call the viruses or mutating or weakening, it's because when it's assembled, it's assembled with broken bits that don't work well, and it's not so much of a fierce virus anymore, and so the virus has mutated or weakened.
All right.
So mostly um, all right, so there's very, very, very few viruses that do the replication process well, except for little tiny viruses where there's very few components, it's not very um uh tricky assembly, and so on.
Cove is a big virus.
SARS-CoV-2 is honking big.
Okay, so SARS-CoV-2 is a honking big virus, and it's got big particles that are complex.
Some of them even have crude error checking.
So some of them that being the case, our honking big virus is difficult for it to do a good job.
And so it turns out that this is why SARS-CoV-2 is not very good on surfaces.
That's because most of the particles that SARS-CoV-2 is going to reproduce, the millions of them that it wants you to spew out and so on, are not going to be infectious because they're bad copies.
So there are all these little screw-ups out here.
And it's what's known as the PFU ratio is very low.
And that is to say it has a very low number of infectious particles versus overall particle production.
Alright?
And so as a as a nasty, it's really nasty if you've got low vitamin D and low vitamin C. But in its general sense of a nasty, you know, it's not bad.
They could have engineered a much more virulent virus particle that could have been smaller, maybe they're working on that.
I don't know.
But there's components that we don't have to deal with on this, in the sense that these virus particles for SARS-CoV-2, being the size they are, are in the and basically it looks to our advantage that uh the really,
really, really wicked uh viruses that are very small that don't have this inbuilt flaw of having um a disproportionate uh um broken bits in their reproductive cycle, uh, are going to be extremely hard to engineer.
And the more they try and cram shit into a virus, the more it's gonna get into the big honking category and have the big honking uh uh lice out of mostly non-infective, not so good, you know, broken virus particles.
And see, this is where it's at.
On surfaces, we're picking up all the broken bits in our tests.
So we can't tell, uh we don't have an effective test that tells us whether or not it's coronavirus, that it's this coronavirus, and that it's an infective particle of this coronavirus, because we can't yet test for completeness of the virus.
All we can test for are its throw-offs, the antigens, and so on, the effects of the uh RNA messengers within the blood, etc.
So uh that being the case, uh it's not it's not an ultimate nasty, you know, it's not the andromeda strain kind of stuff.
It's bad if you've got the underlying conditions that allow it to be bad, but ultimately it's not bad other than that.
And the lockdowns were a big um uh screw up, okay, or they were an op.
Either way, it didn't do anything to aid anybody that was ever going to get it as an infection or perish of it as an infection.
It may have just simply shifted in in time.
Because if your vitamin D weak and you encounter it two years from now, you're gonna die of it if you'd encountered it this year and you were vitamin D weak, you're gonna die of it, right?
So, under the circumstances, um told it's a very dangerous thing, but I'm not worried about it for myself, not worried about it much for my wife, she's getting tested.
We're getting our vitamin D levels up, minus is 63, so I'm well above the category of those people that are reporting with the disease.
Uh, so in spite of my age, I'm not particularly a potential victim for it.
Under the circumstances, uh, as I say, it's not a um, it's not the society killer I thought it would be.
All right, it's devastating to the Chinese.
It's gonna be devastating to those cultures that don't supplement, whether through uh poor diet, through uh economic issues like India, uh these kind of uh things will find it to be a very devastating disease.
There will be many, many, many countries that will have a right to um uh complain loudly that the Chinese let this out on us, uh, all of those people that have died of it, certainly um we have had excess deaths.
It is more strong than the flu.
The excess deaths we've had have been um have may have actually reduced and been absent the flu.
In other words, the hundred and ten thousand people or more that they've said have died of it in the United States, and it's still not done, um uh likely we've reduced the the 70,000 people that would have died of regular flu at this time because of the conditions and under which we'd we'd uh done the social distancing and so on, where uh influenza really does uh transmit effectively this way.
So, under those circumstances, we still have um serious issues coming to us this fall relative to COVID.
It'll be very necessary for all of us to supplement uh continuously for the next few years until we've got this thing eradicated, and that may be five years or more, depending on the um uh how long it takes the developing world to catch up on this.
Uh and we've got other issues that are going to impact all of us.
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